What neurophysiological change occurs post-stroke affecting gait?

Following a stroke, one of the primary neurophysiological changes is the disruption of the corticospinal tract. This pathway is crucial for voluntary motor control and is responsible for transmitting signals from the brain to the spinal cord, which then innervates muscles to produce movement. When a stroke occurs, particularly in regions involved with motor function, it can lead to damage or interruption of this tract.

This disruption results in a variety of motor deficits, including weakness (paresis) and loss of coordination, which significantly impact gait and other movements. Individuals may find it more challenging to initiate movement or maintain control during walking due to this altered neural transmission. The consequence of corticospinal tract disruption typically manifests as impaired ability to control the lower extremities, leading to asymmetrical gait patterns, difficulties in balance, and an increased risk of falls.

In essence, the corticospinal tract's integrity is essential for normal gait function, making its disruption a key factor in understanding post-stroke mobility challenges.